Canine distemper virus

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Canine distemper virus (CDV) is closely related to measles virus. Both share a common ancestor, along with rinderpest and phocine distemper viruses.[1]

From ProMEDMail: Canine distemper virus is the pathogen most frequently causing outbreaks at the carnivore wildlife-domestic interface. As distemper and rabies affect the central nervous system, they both cause behavior disorders. Distemper usually results in stumbling and lack of shyness; animals can seem dumb and/or friendly. Animals showing the same signs might be rabid.

In addition to insuring that your pet is up to date on canine distemper vaccine, also make sure your pet is up to date on its rabies vaccination.

Canine distemper is a contagious and serious disease caused by a virus that attacks the respiratory, gastrointestinal and nervous systems of puppies and dogs.

The virus can also be found in wildlife such as foxes, wolves, coyotes, raccoons, skunks, mink and ferrets and has been reported in lions, tigers, leopards and other wild cats as well as seals.

Puppies and dogs most often become infected through airborne exposure (through sneezing or coughing) to the virus from an infected dog or wild animal. The virus can also be transmitted by shared food and water bowls and equipment. Infected dogs can shed the virus for months, and mother dogs can pass the virus through the placenta to their puppies.

Because canine distemper also impacts wildlife populations, contact between wild animals and domestic dogs can facilitate the spread of the virus. Canine distemper outbreaks in local raccoon populations can signal increased risk for pet dogs in the area.

All dogs are at risk, but puppies younger than 4 months old and dogs that have not been vaccinated against canine distemper are at increased risk of acquiring the disease.

Initially, infected dogs will develop a watery to pus-like discharge from their eyes. They then develop fever, nasal discharge, coughing, lethargy, reduced appetite, and vomiting. As the virus attacks the nervous system, infected dogs develop circling behavior, head tilt, muscle twitches, convulsions with jaw chewing movements, and salivation ("chewing gum fits"), seizures, and partial or complete paralysis. The virus may also cause the footpads to thicken and harden, leading to its nickname "hard pad disease."

In wildlife, infection with canine distemper closely resembles rabies.

Distemper is often fatal, and dogs that survive usually have permanent, irreparable nervous system damage.

Veterinarians diagnose canine distemper through clinical appearance and laboratory testing. There is no cure for canine distemper infection. Treatment typically consists of supportive care and efforts to prevent secondary infections; control of vomiting, diarrhea and neurologic symptoms; and combating dehydration through administration of fluids. Dogs infected with canine distemper must be separated from other dogs to minimize the risk of further infection.

While treatment is non-specific, can be lengthy and sometimes difficult, some animals do survive but may have permanent neurological problems.

Vaccination is crucial in preventing canine distemper.

  • A series of vaccinations is administered to puppies to increase the likelihood of building immunity when the immune system has not yet fully matured.
  • Avoid gaps in the immunization schedule and make sure distemper vaccinations are up to date.
  • Avoid contact with infected animals and wildlife
  • Use caution when socializing puppies or unvaccinated dogs at parks, puppy classes, obedience classes, doggy day care and other places where dogs can congregate.
  • Pet ferrets should be vaccinated against canine distemper using a USDA-approved ferret vaccine.

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It has been speculated that CDV could jump species to humans. A recent conference presentation[2] suggested that this would require very few mutations - the virus can alread infect some primates. The pattern of CDV disease in other mammals suggests that it may be more neuropathogenic than measles - it may cause conditions resembling SSPE; although, of course, this remains speculative. The presentation suggested that widespread immunity to measles (historically through natural infection, and more recently through vaccination) provides cross-immunity to CDV; but that if measles is eradicated, and vaccination consequently ceases, we may become vulnerable to CDV.