Herpes zoster oticus

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Contents

Introduction

In 1907 James Ramsay Hunt suggested that herpes zoster oticus resulted from a geniculate ganglionitis although many regard it as a neuritis or polycranial neuropathy. This cause (latent herpes zoster) of the syndrome is now a specific disease entity[1][2][3]. Treatment is with antivirals such as acyclovir and corticosteroids[4] although there are no randomised controlled trials. Occasionally surgical decompression of the facial nerve is necessary.

Pathology

Inflammatory involvement of the geniculate ganglion and its nearly related vestibulocochlear nerve within the bony facial canal as per James Ramsay Hunt's classical description.

Features

The syndrome has:

  1. Cochleovestibular symptoms
  2. Facial paralysis
    • Usual dense
    • Recovery can be prolonged

and was first described by Letulle in 1882. The additional criteria specific to herpes zoster is

  • Erythematous vesicular rash on the ear (zoster oticus) or in the mouth.

It may be associated with:

  • Paralysis of other cranial nerves.
  • Severe otalgia
  • Ear canal oedema

Due to evolution of signs it may initially be indistinguishable from Bell's palsy. Indeed a proportion of patients with this later condition have herpes zoster rather than herpes simplex as the cause

The facial palsy and 8th nerve symptoms can also be caused by acute otitis media due to herpes simplex and bacteria[5].

Investigations

  • Audiogram (pain permitting)
  • CT scan to exclude abscess formation

Management

  • Antiviral drug :Acyclovir, adult dose is 800mg, five times per day for 7 days
  • Analgesia
  • Splint ear canal with Pope wick
  • Antibiotic/steroid drops reduce otalgia and swelling

References

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