Adermatoglyphia (immigration delay disease) is the absence of finger prints. It is a little challenging for those who use dermatoglyphics for human identification. Genetic causes are rare. SMARCAD1 gene mutations that affect the shorter skin-specific isoform of SWI/SNF-related matrix-associated actin-dependent regulator of chromatin subfamily A containing DEAD/H box 1 have been described as autosomal dominant. It has been reported in dyschromatosis universalis hereditaria (DUH) but in this case the gene responsible has not yet been characterised. Acquired causes through trauma or burns are more common. Patients with Kindler syndrome can acquire it. Capecitabine can cause it.
- ↑ Burger B, Fuchs D, Sprecher E, Itin P. The immigration delay disease: adermatoglyphia-inherited absence of epidermal ridges. Journal of the American Academy of Dermatology. 2011 May; 64(5):974-980.(Print-Electronic) (Link to article – subscription may be required.)
- ↑ Kumar S, Bhoyar P, Mahajan BB. A case of dyschromatosis universalis hereditaria with adermatoglyphia: A rare association. Indian dermatology online journal. 2015 Mar-Apr; 6(2):105-109.(Print) (Link to article – subscription may be required.)
- ↑ Cohen PR. Capecitabine-Associated Loss of Fingerprints: Report of Capecitabine-Induced Adermatoglyphia in Two Women with Breast Cancer and Review of Acquired Dermatoglyphic Absence in Oncology Patients Treated with Capecitabine. Cureus. 2017 Jan; 9(1):e969.(Electronic) (Link to article – subscription may be required.)